By Mariella De Biasi
Nicotine Use in psychological disease and Neurological Disorders, a quantity within the International evaluate of Neurobiology sequence, is a accomplished evaluate of the state of the art examine into nicotine use in psychological disease and neurological issues. It studies present wisdom and figuring out, additionally presents a kick off point for researchers and practitioners coming into the field.
- Provides a huge assurance of topic matter
- Presents a finished assessment of the cutting-edge learn into nicotine use in psychological disorder and neurological disorders
- Ideal for researchers and practitioners, and people simply coming into the field
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Additional resources for Nicotine Use in Mental Illness and Neurological Disorders,
2008; Salas, Pieri, & De Biasi, 2004). , 2007). , 2007). , 2009; Parasuraman, Greenwood, Kumar, & Fossella, 2005). , 1999). , 2003). , 2004). , 2000). Only a handful of studies have addressed the issue of CHRNB2 or CHRNA4 changes in schizophrenia, or the role of polymorphisms in these genes and smoking in patient populations. Linkage analysis has identified the 1q21–22 region, which contains the CHRNB2 gene, in relationship to familial schizophrenia (Brzustowicz, Hodgkinson, Chow, Honer, & Bassett, 2000).
38 Robert E. Featherstone and Steven J. 1 CHRNA7 Two types of nicotine receptor were initially identified based on response to alpha-bungarotoxin and affinity for nicotine, with one receptor type characterized by high alpha-bungarotoxin binding/low nicotine affinity and the other characterized by lack of bungarotoxin binding/high affinity for nicotine (Cordero-Erausquin, Marubio, Klink, & Changeux, 2000; Lindstrom, 1997; Zoli, Lena, Picciotto, & Changeux, 1998). A role for the low nicotine affinity bungarotoxin-binding receptor in sensory gating was first established in rats using intraventricular administration of alpha-bungarotoxin, which was shown to reduce sensory gating (Luntz-Leybman, Bickford, & Freedman, 1992).
2013). , 2013). , 2014). 50 Robert E. Featherstone and Steven J. 3 Disease-Related Symptoms Several rodent studies have assessed the effects of α7 agents on surrogate measures of positive symptoms. , 2005). , 2009). , 2009). To date, there is a lack of studies that have assessed the effects of α7 agents on rodent models of anhedonia. Some evidence suggests that the anxiolytic effects of nicotine may be due in part to activation of α7 receptors. , 2009). , 2014). , 2013). , 2014). The lack of effect of α7 agents on positive symptoms in humans contrasts sharply with positive findings in preclinical rodent models.